Which sounds better to you: taking a physiologic dose of T3, or a heroic bolus that acts like an illicit stimulant? If you get your care from alt med, chances are you’ll be slammed with the latter.
In assessing whether there is value in monitoring T3 levels and restoring them to normal in hypothyroidism, I think it helps to return to first principles. And no, “more T3 is better” is not a first principle.
In this next post in the T3 Controversies series, we cover whether T3 therapy must be used to account for tissues that can’t make their own T3.
In this first post of my “T3 Controversies” series, I address the claim that there are acquired forms of tissue resistance to thyroid hormone, which can be treated with high-dose T3 therapy.
Since the blog’s inception, my philosophy regarding comments has been to allow anyone to say anything. Unfortunately, my permissive approach has allowed a certain type of comment to flourish – the kind that espouses some of the same non-evidence-based claims that this blog was created to debunk. Well, there’s a new sheriff in town.
Americans considering surgery for Hashimoto’s would require a level of rigorous screening that is found mainly in organ transplant and astronaut-training programs. Without that degree of rigor, it is my opinion that most who go under the knife will come out the other side with the same constellation of nonspecific symptoms they had pre-surgically.
Is it time to throw out your thermometer? Here’s an evidence-based look at why BBT is not helpful for diagnosing/managing hypothyroidism.
There is a recurring thread throughout the comments on some of my posts, with respect to people’s desire to order their own blood work. To…
I give up. Like Danny Glover’s iconic character in Lethal Weapon: via GIPHY You win, alt med – everyone is hypothyroid. Lab testing in the…
I’m kicking this post off with a disclosure: what you read today is heavy on speculation and light on evidence because…well…the evidence in this space…